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med64 recording system  (Panasonic Healthcare)


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    Panasonic Healthcare med64 recording system
    Med64 Recording System, supplied by Panasonic Healthcare, used in various techniques. Bioz Stars score: 94/100, based on 104 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/multi-array+system/MED64+Multi-electrode+Array+System/pm42469800-104-41-45
    Average 94 stars, based on 104 article reviews
    med64 recording system - by Bioz Stars, 2026-10
    94/100 stars

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    Related Articles

    other:

    Article Title: Discovery of a β-arrestin-biased CCKBR agonist that blocks CCKBR-dependent long-term potentiation.
    Article Snippet: All the recordings were carried out using the MED64 system (MED, Panasonic Alpha-Med Sciences).

    Article Title: Characterization of excitatory synaptic transmission in the retrosplenial cortex of adult mice
    Article Snippet: A commercial MED64 recording system (Panasonic) was used for extracellular field potential recordings.

    Article Title: Characterization of excitatory synaptic transmission in the retrosplenial cortex of adult mice.
    Article Snippet: A commercial MED64 recording system (Panasonic) was used for extracellular field potential recordings.

    Article Title: Discovery of a β-arrestin-biased CCKBR agonist that blocks CCKBR-dependent long-term potentiation
    Article Snippet: All the recordings were carried out using the MED64 system (MED, Panasonic Alpha-Med Sciences).

    Article Title: GluK1-containing kainate receptor-dependent long-term depression in the anterior cingulate cortex modulates chronic pain.
    Article Snippet: The slices were transferred to a submerged recovery chamber with oxygenated (95% O2 and 5% CO2) ACSF at room temperature for at least 1 h. Preparation of the multielectrode array The multielectrode array system used in the current study was a MED64 recording system (P515A, Panasonic Alpha-Med Sciences).

    Article Title: Biphasic synaptic plasticity of the medial thalamic-cingulate pathway contributes to pain hypersensitivity in vivo
    Article Snippet: Multi-channel fEPSPs were recorded using the MED64 multichannel recording system (Panasonic Alpha-Med Sciences).

    Article Title: Recruitment of cortical silent responses by forskolin in the anterior cingulate cortex of adult mice.
    Article Snippet: Slices were transferred to a submerged recovery chamber with 12 oxygenated (95% O2 and 5% CO2) ACSF at room temperature for at least 1 h. 13 14 Preparation of the multielectrode array 15 A commercial MED64 recording system (Panasonic) was used for extracellular field potential 16 recordings.



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    (A) Representative image of multi-unit recordings setup of ex vivo thalamic horizontal slices in a humidified, oxygenated interface chamber. (B) Schematic of multi-unit activity recordings with a <t>Neuronexus</t> <t>16-channel</t> recording electrode positioned in the thalamus following electrical stimulation of the internal capsule. (C) Representative 3-second recording of intrathalamic multi-unit activity evoked by stimulation of the internal capsule (black dot indicates time of stimulation). Only 12 of 16 channels are shown for clarity. (D) Collapsed activity from 12 recording channels in (C). Black dot indicates time of stimulation. (E) Post-stimulus time histogram of instantaneous spiking frequency from multi-unit activity recorded across 16 channels (all 16 channels were analyzed, 12 representative channels are displayed in (C) from 15 slices from 9 hCSF1 (WT) mice, 8 slices from 7 hCSF1-Grn −/− +KOxMG mice, and 12 slices from 9 hCSF1-Grn −/− +WTxMG mice. The grey box denotes early response (0–1.5 s), and the white box denotes delayed response (1.5–3 s) after stimulation. (F) Enlarged instantaneous frequency of the delayed response (1.5–3 s) from (D). (G) Relative probability of eliciting spiking during the direct response (0–1.5 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (* p = 0.0024). (H) Instantaneous frequency of spiking during the early response (0–1.5 s) after stimulation; Kruskal–Wallis test, H(3) = 6.767, p = 0.0797. (I) Relative probability of eliciting spiking during the delayed response (1.5–3 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (**** p < 0.0001). (J) Instantaneous frequency of spiking during the delayed response (1.5–3 s) after stimulation; Kruskal-Wallis test, H(3)=56.6, **** p < 0.0001, and Dunn’s multiple comparisons test (**** p < 0.0001). Data are presented as mean ± SEM. Comparisons not shown are not significant.
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    Image Search Results


    (A) Representative image of multi-unit recordings setup of ex vivo thalamic horizontal slices in a humidified, oxygenated interface chamber. (B) Schematic of multi-unit activity recordings with a Neuronexus 16-channel recording electrode positioned in the thalamus following electrical stimulation of the internal capsule. (C) Representative 3-second recording of intrathalamic multi-unit activity evoked by stimulation of the internal capsule (black dot indicates time of stimulation). Only 12 of 16 channels are shown for clarity. (D) Collapsed activity from 12 recording channels in (C). Black dot indicates time of stimulation. (E) Post-stimulus time histogram of instantaneous spiking frequency from multi-unit activity recorded across 16 channels (all 16 channels were analyzed, 12 representative channels are displayed in (C) from 15 slices from 9 hCSF1 (WT) mice, 8 slices from 7 hCSF1-Grn −/− +KOxMG mice, and 12 slices from 9 hCSF1-Grn −/− +WTxMG mice. The grey box denotes early response (0–1.5 s), and the white box denotes delayed response (1.5–3 s) after stimulation. (F) Enlarged instantaneous frequency of the delayed response (1.5–3 s) from (D). (G) Relative probability of eliciting spiking during the direct response (0–1.5 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (* p = 0.0024). (H) Instantaneous frequency of spiking during the early response (0–1.5 s) after stimulation; Kruskal–Wallis test, H(3) = 6.767, p = 0.0797. (I) Relative probability of eliciting spiking during the delayed response (1.5–3 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (**** p < 0.0001). (J) Instantaneous frequency of spiking during the delayed response (1.5–3 s) after stimulation; Kruskal-Wallis test, H(3)=56.6, **** p < 0.0001, and Dunn’s multiple comparisons test (**** p < 0.0001). Data are presented as mean ± SEM. Comparisons not shown are not significant.

    Journal: bioRxiv

    Article Title: Transplantation of Human IPSC-derived Microglia Ameliorates Neuropathology and Circuit Dysfunction in Progranulin-Deficient Mice

    doi: 10.64898/2026.01.13.699312

    Figure Lengend Snippet: (A) Representative image of multi-unit recordings setup of ex vivo thalamic horizontal slices in a humidified, oxygenated interface chamber. (B) Schematic of multi-unit activity recordings with a Neuronexus 16-channel recording electrode positioned in the thalamus following electrical stimulation of the internal capsule. (C) Representative 3-second recording of intrathalamic multi-unit activity evoked by stimulation of the internal capsule (black dot indicates time of stimulation). Only 12 of 16 channels are shown for clarity. (D) Collapsed activity from 12 recording channels in (C). Black dot indicates time of stimulation. (E) Post-stimulus time histogram of instantaneous spiking frequency from multi-unit activity recorded across 16 channels (all 16 channels were analyzed, 12 representative channels are displayed in (C) from 15 slices from 9 hCSF1 (WT) mice, 8 slices from 7 hCSF1-Grn −/− +KOxMG mice, and 12 slices from 9 hCSF1-Grn −/− +WTxMG mice. The grey box denotes early response (0–1.5 s), and the white box denotes delayed response (1.5–3 s) after stimulation. (F) Enlarged instantaneous frequency of the delayed response (1.5–3 s) from (D). (G) Relative probability of eliciting spiking during the direct response (0–1.5 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (* p = 0.0024). (H) Instantaneous frequency of spiking during the early response (0–1.5 s) after stimulation; Kruskal–Wallis test, H(3) = 6.767, p = 0.0797. (I) Relative probability of eliciting spiking during the delayed response (1.5–3 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (**** p < 0.0001). (J) Instantaneous frequency of spiking during the delayed response (1.5–3 s) after stimulation; Kruskal-Wallis test, H(3)=56.6, **** p < 0.0001, and Dunn’s multiple comparisons test (**** p < 0.0001). Data are presented as mean ± SEM. Comparisons not shown are not significant.

    Article Snippet: Extracellular multi-unit activity (MUA) recordings were obtained with a linear 16-channel multi-electrode array (Neuronexus) that spanned the nRT and VB thalamic regions.

    Techniques: Ex Vivo, Activity Assay